Arsenic and Old Lace Essay Example for Free

Arsenic and Old Lace Essay The play opens in the living room of the Brewster home, where Abby and Martha Brewster, and their nephew, Teddy live. Teddy, who is rather crazy, believes himself to be Theodore Roosevelt. Mortimer enters and announces to his aunts that he intends to marry Elaine, the minister’s daughter, whom he is taking to a play that evening. Things begin to unravel as Mortimer lifts the lid to the window seat. He discovers the dead body of Mr. Hoskins and assumes that Teddy has killed him. However, Abby and Martha tell Mortimer that it was they who poisoned the man with their homemade elderberry wine and that he is the eleventh (or twelfth) gentleman they have poisoned. Meanwhile Teddy is down in the cellar digging what he believes is the Panama Canal but is in reality a grave. Mortimer and Teddys brother Jonathan arrives. Jonathan is accompanied by Dr. Einstein, a plastic surgeon. Dr. Einstein has operated on Jonathan so that he looks like Boris Karloff, the horror film star. Teddy invites Einstein to join him in the cellar, where he believes he is digging the Panama Canal. Einstein quickly returns and confides to Jonathan that there is a hole large enough to bury Mr. Spenalzo (a man Jonathan recently killed) after everyone goes to bed. Once the lights are out and everyone is supposedly asleep, Teddy goes to the window seat to get Mr. Hoskins. At the same time Jonathan and Einstein go to their car to get Mr. Spenalzo. Both plan on burying the bodies in the cellar. Lights blink on and off, bodies are moved from the window seat to the cellar to the car outside, accusations and threats fly back and forth. Due to the commotion at the house, Officer OHara stops by to make sure all is well. When he is sure that everything is alright, he shifts topics and asks Mortimer of a play he is writing. Just then, the lieutenant bursts in and recognizes Jonathan as an escapee from a prison for the criminally insane. Jonathan tells the officers about the bodies in the cellar, but they dont believe him and take him off to prison. Einstein gets away, and Teddy is certified insane and taken to the Happy Dale Sanitarium. Mortimer happily agrees when his aunts insist on going to Happy Dale with their nephew. The aunts then kindly inform Mortimer that he is actually not a member of the Brewster family. He was an illegitimate child and thus can marry Elaine without fear of passing the Brewster insanity on to his children. Mortimer happily departs, but before the women leave their house, they offer a drink to the head of Happy Dale, Mr. Witherspoon. Witherspoon is a lonely older gentleman, and he gladly accepts a glass of the spiked elderberry wine.

Divorce is An American Family Tragedy Essay -- Informative Essays Fami

In America today, one of our main life goals is to marry the person we fall in love with, live happily ever after, and skip gleefully away to live the American dream. In most cases, after marriage then comes children which starts a family. This has been a part of human nature since the beginning. Marriage and family are the backbone of our culture. Families need each other for support, dependence, learning, love, encouragement, and ultimately survival. Parents are the ones that supply these needs, meanwhile supplying their own needs by depending on each other for love and support. Only the two of them can give this support because of what they are to each other, husband and wife. When two people get married, they are obviously in love and feel that they want to spend the rest of their lives with each other. They make the ultimate commitment to love one another and one another only, forsaking all others til death do they part. So if this is such an important part of our culture and we have such high goals to get married, then why do 40% of marriages end in divorce? Why does America have the highest divorce rate in the world? What makes couple who previously had fallen madly in love fall out of love and into divorce? What are the main causes of divorce and what are some of the warning signs that you, if you are married or soon to be married should be award of if you do not want your marriage to fail and become one of the statistics? There are many reasons people divorce and there are always very unique circumstances around certain divorces. According to the U.S. Bureau of the Census taken in 1992, younger people are marrying for the first time and only about 50-60% of these couples? marriages are surviving. That... ... of Denver and PREP Inc. Adults and children are at increased risk for mental and physical problems due to marital distress and divorce. Marital problems are associated with decreased work productivity, especially for men. Married men and women in all age groups are less likely to be limited in activity due to illness than single, separated, or divorced individuals (National Center for Health Statistics, 1997). Also children living with a single parent or adult report a higher percentage of activity limitation and higher rates of disability. They are also more likely to be in poorer health than children who do not come from a divorced family. Think about this before you untie the knot. The dangers that financial problems, lack of communication, and failure to work together as a team can impose on your marriage or future marriage can have devastating effects.

Food Inc Movie Monsanto Essay

Food, Inc. reveals the shocking truths about the food and food chain that America eats. It narrates the industrial production of meat (chicken, beef, and pork), grains and vegetables (primarily corn and soybeans) and lastly about the major food corporations involved. The movie makes us aware about the food that we eat, the hidden politics about the production systems and economic and legal power that the food companies possess. As the film advances, it demonstrates the rise of current method of raw food production as a response to the growth of the fast food industry of the 50’s as compared to traditional methods. It makes a strong point by highlighting that a few multinational corporations who dominate the modern food chain and its production have skewed interests. The emphasis of these food corporations like Monsanto is on business and profitability rather than food quality or health and safety – of the food itself, of the animals produced themselves, of the workers on the assembly lines, and of the consumers actually eating the food. Production of large quantities of food at low direct inputs (most often subsidized) result in enormous profits, which in turn marks greater domination of the global supply of food sources by selected few food corporations. The government conveniently overlooks the power exercised by the food companies in pursuit of cheaper food regardless the negative repercussions. Regulatory agencies, USDA and FDA, sparing from the public eye give their consents to the food corporations such as Monsanto, to manipulate with the public health in exchange of bigger returns. The government approved bigger-breasted chickens, the perfect pork chop, insecticide-resistant soybean seeds, tomatoes that will not go bad are the negative gifts presented by these corporations. Companies like Monsanto throw more science at the problems to bandage the issues instead of uprooting the problem. As a result, new strains of E. coli- the harmful bacteria that causes illness for an estimated 73,000 Americans annually have emerged due to the inappropriate business behavior of the food corporations. The increasing obesity levels, particularly among children, and an epidemic level of diabetes among adults is a gift from the food corporations.

Modern Ideas for Treating Epilepsy

IntroductionEpilepsy is a chronic disorder characterized by recurrent unprovoked seizures, and is associated with a variety of medical conditions and neurological diseases. â€Å"Antiepileptic medications attempt to treat this chronic seizure propensity, and, by definition, antiepileptogenic drugs aim to prevent the natural history of the epileptic disease† (Raman, Rho, & Cavazos, 2004).Epileptic seizures are paroxysmal clinical events arising from neuronal hyperexcitability and hypersynchrony of the cerebral cortex, either locally or simultaneously in both hemispheres. A seizure occurs when there is a sudden imbalance between the excitatory and inhibitory inputs to a network of neurons such that there is overall excessive excitability.The behavioral manifestations of a seizure depend on the area of the area of the cerebral cortex that is involved – directly, in the seizure focus, or indirectly, through recruitment and propagation of this abnormal paroxysmal neuronal ac tivity. Convulsions are defined as seizure that includes motor manifestations such as repeated and rhythmic jerking of the limbs, most often due to involvement of the motor cortex. Anticonvulsant drugs are medications that attempt to compensate for this abnormal cellular hyperexcitability by shifting the delicate balance back toward its normal state.Treatment for EpilepsyProspective population-based studies have demonstrated that 65-70 per cent of patients attain at least a 5-year remission and that half of these will successfully stop treatment. Thus, the â€Å"long term prognosis of epilepsy is good for most patients with a short history of seizures† (Appleton, 2001). The question arises as to when treatment should be started and when it can be stopped. Prophylactic treatment may be undertaken in patients with a high prospective risk of epilepsy after head injury and craniotomy for various neurosurgical conditions, although no evidence exists that antiepileptic treatment is effective in such cases.â€Å"The main form of treatment for epilepsy is drugs† (Scambler, 1989). The majority of people can be rendered seizure-free by pharmacological mean, although chronic intractable epilepsy develops in approximately 20 per cent of cases. Occasionally surgery may be appropriate for those who have a single discrete focal abnormality and whose seizures have been unresponsive to drug therapy, but the proportion of those with epilepsy likely to benefit from surgery will probably remain small. Sometimes people can learn to control their seizures by avoiding clear precipitants, for example alcohol or lack of sleep. But the large majority requires anti-convulsant medication, often over a period of many years.Five drugs are commonly used for the management of partial and grand mal seizures: phenytoin, carbamazepine, sodium valporate, phenobarbitone and primidone. All show similar therapeutic results, but phenobarbitone and primidone tend to be used less because of their sedative effects.  Unfortunately, other studies have shown the â€Å"high incidence of behavioral side-effects from the treatment† (Là ¼ders, 2001), an important issue when weighing the pros and cons of continuous therapy.These side-effects occur in 40% of treated children and include irritability, hyperactivity, sleep pattern disturbances, and the possibility of diminished intelligence, but its effectiveness has now been questioned. Mephobarbital has the same side effects as Phenobarbital and phenytoin and carbamazepine do not appear to be effective. The potential role for newer antiepileptic drugs such as lamotrigine and gabapentin is unknown.  The Consensus Development Conference on Febrile Seizures attempted to make the best possible recommendations regarding treatment. The panel recommended ‘considering’ continuous anticonvulsant prophylaxis only in the presence of any known high-risk factors. In this basis, only a small percentage of children would require treatment.References:Appleton, R. (2001). Epilepsy (4th ed.). Massachusetts, USA: Informa Health Care.Là ¼ders, H. (2001). Epilepsy: Comprehensive Review and Case Discussions. Great Britain: Informa Health Care.Raman, S., Rho, J. M., & Cavazos, J. E. (2004). Epilepsy: Scientific Foundations of Clinical Practice. New York – Basel: Informa Health Care.Scambler, G. (1989). Epilepsy. New Fetter Lane, London: Routledge.

Agoraphobia Disorder or Panic Attack Essay

Agoraphobia Disorder or Panic Attack Essay Agoraphobia Disorder Essay Example Agoraphobia Disorder Essay Example Agoraphobia is a disease that is closely connected with the Panic Disorder. People with such disease are afraid to go into places or events where they have experienced panic or stress attack and anticipate it to happen again. Usually, a person who suffers from agoraphobia avoids visiting public places because he/she feels a need to escape or expects to be physically offended. Shopping centers, public transport and sports areas are the taboo places, which sick people refuse to visit. This paper discusses the clinical evolutionary learning of agoraphobia, its relation to PD and PA and causes of AG development. This paper explores six historical and social background publications that report on results from explorations conducted on different indications and classifications of agoraphobia disorder. However, the paper varies in DSM characteristics and AG relation to PA and PD neurosis. Wittchen et al. (2010) mentioned that only 23,5% of people suffering from PA developed agoraphobia and 50% developed PD. Despite various attempts to specify the features of agoraphobia, the assumptions still exclude each other. This paper discusses the clinical evolutional learning of agoraphobia, its relation to PD and PA and the contextual influences on AG development. Agoraphobia in Revision of the DSM and ICD The mid-80s were distinct by performed striking differences, such as self-rating tools, unstructured system and diagnostic without symptomatic focus. Panic attacks were directly associated with various disorders (substance and mood disorders, anxiety are actually not key identifications of agoraphobia or panic disorder). In addition, here is no large risk for PD or PA to be a result of agoraphobia disorder. According to DSM-IV-TR, only 2,4% of sick people developed PD and 11,6% developed PA. (Wittchen et al., 2010, p. 118). A number of issues caused medical interest towards fears and phobias, including agoraphobia. First, it is connected with the high percentage of agoraphobia disorder among others forms of phobias. Second, the structural composition of the phobias is not constant. It is noticeable along with the affective, sensory, vegetative and dietary components. Third, agoraphobia is a disease that hardly passes without any treatment. The disease is more inclined to progression and needs a long-term treatment. The first time agoraphobia was mentioned in 1871 (Westphal’s classical description) as a paradigm for nervous disorders. Until the introduction of DSM-III-R, AG was identified in the medical literature as a regular phobia or neurosis. In 1970s agoraphobia was codified as a distinctive syndrome of multiple fears (ICD-9) and it retains the same codification today (ICD-10). In the USA, the agoraphobia within DSM system was considered a result of subdividing phobic nervous and anxiety disorder (DSM-III). The DSM-III has such symptoms as fear of being alone, avoidance of public places or panic attack when being there (especially under assumption of unavailable help in case of sudden incapacitations). In fact, the DSM definition is not very different from other learned disorders and the ICD explanation of agoraphobia, which is one of the disadvantages of the system. In 1980, agoraphobia was rather considered a form of panic attack (PA) than a special form of phobia. It was explained by temporary panic attacks on the initial phase of agoraphobia development and diagnostics omissions. The person developed an increased anticipatory fear of having panic attack and, therefore, in different ways tried to escape or avoid the indicators that cause them. The diagnosis of AG without panic attacks was made when the history of panic attacks was lost. Beginning from DSM-III-R, AG was described as a typical response to cases when PA had occurred. In addition, the AG in DSM-III-R was seen exclusively as a secondary complication and it was attached to panic attacks and panic disorders as an opposition to popular clinical and experimental achievements. According to Wittchen et al., â€Å"with consecutive DSM revision, the residual status of AG within the construct of PA and PD has been increasingly more pronounced† (2010, p. 115). DSM-IV-TR recognizes agoraphobia as impossible to code disorder. Alternatively, the panic disorder with agoraphobia or agoraphobia without the history of PD was provided within code disorders of agoraphobia similarity. The DSM-IV-TR is represented by complex differential diagnostic description with important considerations. It restricts the disease to people, who have AG related to fear of PD symptoms (e.g. diarrhea or dizziness). Thus, the specific diagnosis prescription was based on the definition o f two syndromes, including panic attacks (a complex of mental disorders) and agoraphobia (as a part of panic disorder or AG without the history of PD). Additionally, the discrepancy between DSM and ICD increased. The ICD-9 and ICD-10 retained agoraphobia as a separate disorder, not a form of PA or PD. The DSM-IV-TR defines agoraphobia in a different way than it was done before. In classical variations, the diagnosis was tied to PA or PD concepts. The DSM-IV-TR defined panic attack as a symptom. According to Barlow, â€Å"this conceptual development was based mainly on the observation in some studies, which use DSM-III-R criteria, AG patients without PA or extremely panic-rare features† (Barlow, 2002, p. 30). Thus, the assessment instruments and diagnostic criteria suspect the opinion that agoraphobia cannot be diagnosed without the context of panic-like symptoms or primary panic attacks. The implicit hierarchical DSM-IV made impossible to create systematic scheme as a productive solution, since the different diagnostic interviews and two discrepant medical criteria regress the treatment. It is important to mention that both panic attacks and PD are comorbid with agoraphobia. Some AG patients may develop their disease under the influence of panic-like symptoms and PA. On the other hand, the agoraphobia is an independent disease, the PA and PD are causally linked to agoraphobia and the clinical utility diagnoses AG as a separate disease. In addition, the researches on how to specify the explicit criteria to agoraphobia continue. One of the treatment disadvantages is that there is an undefined solution of how to diagnose patients whose DSM-IV-TR required symptoms of PD or PA were not noticed. There is a discussion whether in such cases it is better to diagnose NOS (anxiety disorder) or a DSM phobia. The general definition of the agoraphobia needs more specific characterization â€Å"and cues beyond the occurrence or fear of panic-like symptoms† like phobias (Wittchen et al., 2010, p. 115). A mistaken omission makes it impossible to specify mandatory criteria. For instance, when a person recognizes that his fear is unreasonable, excessive and notices the exposure cases and impairment symptoms. A characteristic cluster must be defined when the agoraphobia syndrome covers two to four prototypical situations (as stipulated in the ICD-10 demand). DSM-IV-TR has no such characteristic that is why the DSM studies define the diagnosis without constant compulsory symptoms (Social or Specific Phobia). Overall, the DSM-III modified the diagnostic qualification of the agoraphobia. It was critically reexamined as overinclusive, since it did not restrict agoraphobia to obvious avoidance behavior. In DSM-III-R, agoraphobia was diagnosed, when a person experienced anxiety about having panic attacks, and avoided to be alone or in distress. Nevertheless, this idea can be neglected, because a person can travel alone despite the need of having somebody near. In addition, the classification of AG levels was omitted, including none, mild, moderate, severe. DSM-IV â€Å"is no longer the case with situational avoidance of equal footing with distress and use of companions† in establishment of the dichotomous diagnosis of agoraphobia (whether it is present or absent) (APA, 2000, p. 18). Schmidt and Cromer (2008, p. 161) criticized it, because the reduction of the agoraphobia specification from 4-point scale to present/absent dichotomous means that the last one is superior and has better organized assessment of phobias. As a result, the clinical utility and predictive value decreased. Thus, both opinions left the issue unresolved. One of the advantages is that now there are few options outline the key positions on agoraphobia in DSM-V. First, agoraphobia must be excluded as a classification diagnosis and become an additional part of PD. Second, AG must be recognized as a specific phobia. Then, the existing diagnostic categories must retain panic disorder without agoraphobia, panic disorder with agoraphobia, and agoraphobia without the history of PD. Finally, there must be more explicit diagnostic criterion for agoraphobia as a separate category. Clinical Evidence Since 1980s, the global medical explorations concentrated on the modified examination of panic disorder, panic attack and agoraphobia across the world. The criteria were chosen by the models of DSM-III, III-R and DSM-IV. The result demonstrated a spontaneous tendency that rates of agoraphobia without PD are higher than the ones with panic disorder. This factor includes both children and adults. In addition, it was found out that more than a half of the people who suffer from agoraphobia have no panic attacks. The studies had various amount of criteria, which caused assessment and methodological variations, but did not provide a definition of the true differences (like space and cultural influences). Thus, the studies of 1980’s (Diagnostic Interview Schedule – DIS, when even one AG case defined the diagnosis, were changed by the CIDI (Composite International Diagnostic Interview) were influential on the understanding of the agoraphobia. CIDI requires more than the case f or DSM-IV-R criteria, which caused the AG decrease by half and revealed less cases of panic disorder with agoraphobia. The USA studies demonstrate moderation of clinical settings, which has positive progressive impact on further treatment. The clinical practice rarely meets the cases of agoraphobia without the history of PD. C. Faravelli defined eight clinical studies, â€Å"seven with low sample sizes, citing four studies with not a single case of AG without panic and four studies reporting 2-31% of PA among AG patients† (Wittchen et al., 2010, p. 117). PA, PD and AG Temporal Relationship Some studies attempted to define whether agoraphobia has constant relation to spontaneous panic attacks or panic-like symptoms. The clinical and epidemiological experiments and observations demonstrated no evidence for this assumption. The major amount of agoraphobics never experienced panic-like symptoms, PD or any other type of neurotic diseases that preceded the onset of AG. Furthermore, clinical retrospective studies used sensory methods to find prior clinical symptomatology. As a result, before the first panic attack more than a half of patients with agoraphobia had prodromal symptoms such as general anxiety and hypochondriasis. In addition, the research found considerable degree of discrepancy that failed. Nevertheless, it was observed that in up to 50% of all suffering from the AG, PA precedes agoraphobia, â€Å"providing some support for the assumed aetiopathogenic pathway implied in DSM-IV-R† (Wittchen et al., 2010, p. 118). In fact, the concept of panic-agoraphobia spectrum was not taken into consideration with further assuming of reciprocal connection (this aspect is not supported by epidemiological evidence). In advance, the prospective clinic investigations rarely can succeed. One of them described systematical symptoms of agoraphobia, panic disorder and panic attacks. Critical Methodology The evidence that panic-like symptoms and panic attacks frequently play an important pathogenic role in agoraphobia progression was not supported by epidemiological studies. The new methodological grounds proved that the diagnostic interviews were not valid. The experts suppose that those diagnostic tools that are based on background observations that agoraphobia without panic attack and panic disorder is rarely a priori in clinical samples are not able to assess panic issues with sufficient accuracy. Since 1900, a few publications partly neglected CIDI criteria. The main idea of the new algorithmic is requirement of minimum two reported situations before AG diagnosis prescription. A smaller amount of such cases is classified as phobia NOS. As a result of such modification, the general rates of agoraphobia, panic arracks and panic disorders were substantially reduced. Despite such methodological appraisals are considerably sophisticated, some researchers believe that there are omissions in this conception. Age, Gender and Socio-Demographic Issues There is no systematized and generalized description of age and gender difference between agoraphobia and panic disorder. There are few differences between such characteristics of PD and PA with and without agoraphobia. Female preponderance within AG without panic attacks was higher than for panic disorder (American Psychiatric Association, n. d.). The retrospective cross-sectional studies made a conclusion that two thirds of all panic disorder cases appear before 35 years old. There is a rare substantial incidence risk in childhood and adolescence. There are differences between PD with agoraphobia and AG without panic attacks, but there is no notable difference between agoraphobia and panic disorder under the age and gender context. It was noticed, that people who suffer from agoraphobia without panic disorder and PD/AG in most cases have no jobs or they are disable. This conclusion allows assuming that agoraphobia is more widely spread in developed countries with busy and stressful life styles. Thus, a powerful impact of stress a person can experience due to some situations has a very strong influence on nervous system with harsh consequences. Genetic and Familiar Factors The anxiety neurosis and panic disorders are different and, thus, the genetic factors cannot be always an indicator of the possible disease within defined family. Nevertheless, the parental history of agoraphobia and panic disorders can take the core role in further development of panic symptoms in the next generations. Moreover, it can cause development of other diseases or disorders that can be harder to treat due to the factors mentioned above. During the last few decades of medical investigations, it was notified that the higher risk of blood relatives to become AG patients is not confined to agoraphobia. There is a gap in differential algorithm of familial aggregation of agoraphobia and PA/PD. Additionally, the risk for panic disorder might be higher than for agoraphobia. Moreover, agoraphobia and liability of panic attacks cannot be suggested to be on one AG-panic continuum, since agoraphobia is not closely associated with PD aggregation. In more simple way, without any symptoms of PD, agoraphobia cannot be revealed. On the other hand, any parental panic disorder or hard agoraphobia increases the risk to develop anxiety symptoms in offspring. Wittchen et al. (2010) estimated heritability in 61% for agoraphobia and 43-48% for panic disorder (p. 121). The female disorders are more frequent due to possible physiological and hormonal peculiarity, which causes higher heritability. AG Treatment Few studies examined possible opportunities for optimized and effective treatment of all three conditions. The adequate treatment is a financial topic for discussion, because only 42, 1% of agoraphobia diagnosed without PD and 41, 2% of suffering from PD gets enough help. In addition, there are obvious differences between AG without PD patients and PD group, who are not supported by the governmental health care system and psychiatric treatment. Moreover, the panic disorders are more frequent than agoraphobia, which also influences patients to seek for different professionals, since each disease has different complications and characteristics. Furthermore, the situation depends on type and methods of the health care system. For example, the USA and Germany created a structure in a way that the psychotherapists care about AG without PA treatment, whereas PD is a specialization of psychiatrists. Panic disorder and panic attack rarely exist in pure forms. Both are closely associated with other common or somehow related diseases, such as mood, substance and somatoform disorders. In the context of possible transitions from one disorder to another, it was noticed that PAs are strongly connected with psychopathology, but not necessarily characteristic to PD, agoraphobia or other neurotic disorders. Moreover, some commonwealths were found to use substances for psychotic and anxiety disorders. In opposition, agoraphobia is rather connected with the depression and highest probability of anxiety neurosis. Thus, with regard to the issues within PD without AG, AG and PD, the epidemiological studies demonstrate the conclusion that agoraphobia (without panic attacks) and panic-like disorders reveal common disability findings. The most impairing and frequent conditions are PD/AG. There is little evidence of specific differences between agoraphobia without PD and PA. The special observations demonstrated that agoraphobia rather belongs to phobias category, and PA and PD are comorbid disorders. Previously, the treatment was general: there were typical clinical courses of establishments of the space limited type for those who were not able to get better. Sometimes such people were considered as the ones mentally sick who only pretend of being scared. In contrast, the clinical agoraphobia treatment is considered as persistent (SSRI, Benzodiazepines). The clinical agoraphobia treatment is considered as persistent. First, the treatment includes antidepressants (both short and long-term usage). Another important issue is connected with the treatment controversy, because some meta-analysis is biased to CBT alone rather than to pharmacotherapy. The domestic treatment of agoraphobia can be also used to treat PD with AG. In some medical traditions, it was found that pharmacological placebo is significantly more effective than relaxation or alprazolam. The exposure treatment of agoraphobia causes subsequent escape from panic and amelioration. Thus, the pharmacological tools are lacki ng in panic or AG neurosis treatment. Alternatively, it can be reduced by psychotherapeutic cognitive approaches. Agoraphobia developed among many famous people. For example, Horace Leonard Gold (a science fiction writer and editor) had a harsh form. After his wartime trauma, his disease did not allow him to leave an apartment for two decades. Only after hard non-traditional treatment he succeeded. Another person Brian Wilson (singer) also had AG and schizophrenia, but his treatment did not give any result. Those examples demonstrate that this disease can attack anybody and there is no guarantee it will be treated. Axis Issues The Axis model includes five parts that in complex provide a comprehensive diagnosis with full description of the symptoms and factors that affect mental condition. The Axis I describes general depressive/nervous disorder (major characteristics and observation). The Axis II is dedicated to identifying the frequency of disorder attacks. The third Axis describes physical problems that may be related to mental disorders and worsen the condition of the patient (panic, trauma or physical violation). The Axis IV is connected with any kind of threats and dangers (for example, a job loss). The Axis V estimates an ability to function in every fay life (occupational, social and psychological accommodation). Speaking about the multiaxial distinction of Axis I-III, there is no big difference in conceptualization. Physical or biological factors are not related to phobias or neurotic disorders. In addition, medical condition does not directly depend on physical peculiarities. Sometimes they can be controversial, a high-rated diagnosis result of the body functioning does not guarantee impossibility of mental disorder development. Practically, it can be observed in the information provided by the American Psychiatric Association. This establishment monitors the facts, treatment process and results of the AG diagnosis and treatment programs. An important issue is that the conception represents various groups of people whose disorder progressed (by age, gender, surrounding, occupation). For example, one of the hardest forms of agoraphobia was developed in Rita Clark’s social escape. The treatment included complex Axis diagnostics and after 20 years of panic and fear, a woman returned to normal life (American Psychiatric Association, n. d., n. p.). Agoraphobia is an independent form of phobia, characterized by frequent panic attacks, when a person appears in an uncomfortable surrounding. The DSM conceptions tried to conduct a constant algorithm of the symptoms and consequences of agoraphobia disorder. The studying process caused various discussions about the psychological relation of AG to PA or PD. Meanwhile, five phases of Axis were formulated, which allowed providing full diagnosis of the patient simultaneously. The treatment analysis demonstrated that the treatment must include both medical and psychological methods.

Pythagoras of Samos Biography

Pythagoras of Samos Biography Pythagoras, a Greek mathematician and philosopher, is best known for his work developing and proving the theorem of geometry that bears his name. Most students remember it as follows: the square of the hypotenuse is equal to the sum of the squares of the other two sides. Its written as: a 2 b2 c2. Early Life Pythagoras was born on the island of Samos, off the coast of Asia Minor (what is now mostly Turkey), about 569 BCE. Not much is known of his early life. There is evidence that he was well educated, and learned to read and play the lyre. As a youth, he may have visited Miletus in his late teenage years to study with the philosopher Thales, who was a very old man, Thaless student, Anaximander was giving lectures on Miletus and quite possibly, Pythagoras attended these lectures. Anaximander took a great interest in geometry and cosmology, which influenced the young Pythagoras. Odyssey to Egypt The next phase of Pythagorass life is a bit confusing. He went to Egypt for some time and visited, or at least tried to visit, many of the temples. When he visited Diospolis, he was accepted into the priesthood after completing the rites necessary for admission. There, he continued his education, especially in mathematics and geometry. From Egypt in Chains Ten years after Pythagoras arrived in Egypt, relations with Samos fell apart. During their war, Egypt lost and Pythagoras was taken as a prisoner to Babylon. He wasnt  treated as a prisoner of war as we would consider it today. Instead, he continued his education in mathematics and music and delved into the teachings of the priests, learning their sacred rites. He became extremely proficient in his studies of mathematics and sciences as taught by the Babylonians. A Return Home Followed by Departure Pythagoras eventually returned to Samos, then went to Crete to study their legal system for a short time. In Samos, he founded a school called the Semicircle. In  about 518 BCE, he  founded another school in Croton (now known as Crotone, in southern Italy). With Pythagoras at the head, Croton maintained an inner circle of followers known as mathematikoi (priests of mathematics). These mathematikoi lived permanently within the society, were allowed no personal possessions and were strict vegetarians. They received training only from Pythagoras, following very strict rules.  The next layer of the society was called the akousmatics. They lived in their own houses and only came to the society during the day.  The society contained both men and women.   The Pythagoreans were a highly secretive group, keeping their work out of public discourse. Their interests lay not just in math and natural philosophy, but also in metaphysics and religion. He and his inner circle believed that souls migrated after death into the bodies of other beings. They thought that animals could contain human souls. As a result, they saw eating animals as cannibalism.   Contributions Most scholars know that Pythagoras and his followers didnt study mathematics for the same reasons as people do today. For them, numbers had a spiritual meaning. Pythagoras taught that all things are numbers and saw mathematical relationships in nature, art, and music. There are a number of theorems attributed to Pythagoras, or at least to his society, but the most famous one,  the Pythagorean theorem, may not be entirely his invention. Apparently, the Babylonians had realized the relationships between the sides of a right triangle more than a thousand years before Pythagoras learned about it. However, he spent a great deal of time working on a proof of the theorem.   Besides his contributions to mathematics, Pythagorass work was essential to astronomy. He felt the sphere was the perfect shape. He also realized the orbit of the Moon was inclined to Earths equator, and deduced that the evening star (Venus) was the same as the morning star. His work influenced later astronomers such as Ptolemy and Johannes Kepler (who formulated the laws of planetary motion). Final Flight   During the later years of the society, it came into conflict with supporters of democracy. Pythagoras denounced the idea, which resulted in attacks against his group. Around 508 BCE, Cylon, a Croton noble attacked the Pythagorean Society and vowed to destroy it. He and his followers persecuted the group, and Pythagoras fled to Metapontum. Some accounts claim that he committed suicide. Others say that Pythagoras returned to Croton a short time later since the society was not wiped out and continued for some years. Pythagoras may have lived at least beyond 480 BCE, possibly to age 100. There are conflicting reports of both his birth and death dates. Some sources think he was born in 570 BCE and died in 490 BCE.   Pythagoras Fast Facts Born: ~569 BCE on SamosDied: ~475 BCEParents: Mnesarchus (father), Pythias (mother)Education:   Thales, AnaximanderKey Accomplishments:  first mathematician Sources Britannica: Pythagoras-Greek Philosopher and MathematicianUniversity of St. Matthews: Pythagoras BiographyWikipedia Edited by Carolyn Collins Petersen.

Marketing Mix of Domino's pizza Case Study Example | Topics and Well Written Essays - 1500 words

Marketing Mix of Domino's pizza - Case Study Example This research will begin with the statement that in the United Kingdom, the number of the fast food and home delivery restaurants including McDonalds, KFC AND Domino’s Pizza continues to increase rapidly as is the trend for the past five years. The restaurant's explosion in the high-street outlets represents the lucrative feature of the industry, constituting the resulting large amount of competition in the market. The increased competition is a factor notable as a cause for the increased value of sales as experienced by the various participants in the industry. This implies that the consumer has a better access to the restaurants, as they facilitate the services to the customers. However, recent developments in the industry, entailing the increased negative publicity in the market presents a remarkable challenge to the success of the operators. Consequently, the restaurants sought to dispel these trials in the market. Thus, Domino’s Pizza as a leading operator in the m arket, sought the establishment of strategic procedures to counter the issue. Further, their strategy also sought to engage the customers accordingly, increasing the revenue from sales, as well as, facilitating the expansive operations of the business, taking it to the top in the leading operators in the market. This procedural engagement by Domino’s Pizza entailed the establishment of 7P’s of the marketing mix, in their procedural engagement of the customers and establishment of services.